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Stiffness is called the specific pain or discomfort ostavashta tce from 24 to 76 hours after intense exercise a given muscle or muscular group. Once it has been considered that the stiffness is due to the synthesis of lactic acid, but more modern theories suggest that due to the tiny ruptures in muscle tissue, caused by uneven load or abnormal. tce The reason for dropping the lactic acid theory is that after the lactic acid dissipates relatively quickly in the body, it can not explain tce the pain stay the next day. Lactic acid itself as "irritating" microtrauma caused by fitness tce workout and it gives pryava muscle fever!
Although the exact cause is not known, it appears that the type muscular effort is a major factor for the development of muscle strain. Exercises requiring very uneven tce loads (such as running on a slope) would lead to the most severe forms of muscle strain, as such load would cause greater damage to muscle tissue than standard uniform load (eg climbing).
One study claims that the stiffness is not caused by pain in the damaged muscle tissue, and due to the reinforcement process. The muscle reacted workout being reinforced to a degree higher than the previous one by the construction of new segments in the muscle tissue. This process of reinforcement causes cells to grow and put pressure on nearby nerves and arteries, causing muscle strain.
Lactic acid (IUPAC systematic name: 2-hydroxypropan acid) that causes irritation in stiffness is a chemical compound that is involved in several tce biochemical processes. It is a carboxylic acid having the empirical formula C3H6O3. Lactic acid has a hydroxyl group adjacent to the carboxylic acid, which makes it an alpha-hydroxy acid. In solution, it may lose a proton from the acid group, and a lactate ion obtain CH3CH (OH) COO?.
Lactic acid is a chiral and has two optical isomers. One is known as L-(+)-lactic acid or (S)-lactic acid, and the other, its mirror image, is D-(-)-lactic acid or (R)-lactic acid. Biologically important isomer is L-(+)-lactic acid.
L-lactate was prepared by using the enzyme pyruvate lactate dehydrogenase (LDH) in the fermentation process. tce Lactate produced psotoyanno during normal metabolism and physical activity, but does not increase the concentration until the rate of production of lactate did not exceed the rate of its destruction. The rate of destruction szavisi on several factors, tce among which monokarboksilatnite conveyors, and the concentration of LDH isoform and the oxidative capacity of the tissue. The concentration of lactate in the blood is typically 1-2 mmol / L in the resting state, but may increase to 20 mmol / L at the time of vigorous physical activity.
Increases in lactate concentration usually occur in conditions that require tce tissue energy can not be produced by aerobic respiration, ie tissues can not process tce oxygen and substrates quickly enough. Under such conditions, pyruvate dehydrogenase can convert pyruvate to acetyl-coenzyme A sufficiently rapid and starts to accumulate pyruvate. This would normally inhibited glycolysis and reduced production of ATP, if not decreased LDH pyruvate, converting it to lactate in the reaction:
The cumulative tce lactate may be destroyed in several ways: the oxidation of pyruvate to a well oxygenated muscle cells, which is then directly used for the powering of the Krebs cycle, and the conversion in a cycle of glyukoaza Corey the liver via the process of gluconeogenesis.
Whitey Christ tce is a fitness trainer, nutritionist and suplementarist. It deals entirely tce with original tce projects requires the latest ideas and concepts in the field of fitness training, nutrition and nutritional supplements in weight loss programs, increase muscle mass or just for health, tone and a better standard of living. Visit his shop address www.belchohristov.net!
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